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Joint Immobilization

Editor: Kathleen McMahon Updated: 2/15/2026 1:54:20 PM

Introduction

Traumatic musculoskeletal injuries represent a major global and national public health burden and are a leading cause of emergency department utilization. Results from large epidemiologic studies from the United States and worldwide demonstrate that traumatic injuries account for tens of millions of emergency department visits annually, with musculoskeletal trauma contributing substantially to disability, cost, and loss of function across age groups.[1][2] Within this broad category, joint injuries are among the most frequently encountered patterns and are a core focus of both emergency medical services (EMS) and hospital-based trauma systems.[1][3]

In the prehospital environment, joint injuries are often challenging to characterize and appropriately stabilize because of limited diagnostic resources, variable patient factors, and the presence of distracting injuries. Nevertheless, early recognition and structured assessment of suspected joint injuries are essential to guide immobilization decisions and prevent secondary harm during transport.[3][4] These injuries arise from a wide range of mechanisms, including high-energy motor vehicle collisions, pedestrian and motorcycle crashes, sports trauma, and falls from height, and even seemingly minor events such as ground-level falls or low-energy torsional injuries in susceptible individuals. These diverse mechanisms produce patterns ranging from simple sprains to unstable complex fracture-dislocations and periarticular fractures. Because of this spectrum of severity, all suspected joint and extremity injuries in the field should be managed using a standardized strategy that prioritizes hemorrhage control, pain management, anatomic realignment, and immobilization spanning the joints above and below the injury.[2][3]

Timely and well-performed immobilization in the prehospital setting reduces pain, limits further skeletal and soft-tissue damage, decreases blood loss, and protects adjacent neurovascular structures. This may lower the risk of complications such as compartment syndrome and the need for more complex operative reconstruction.[2][3] Observational data from multiple trauma systems have shown that immobilization is frequently performed suboptimally, with errors in splint selection, application, and documentation of distal perfusion and neurologic status. In a series, fewer than 10% of patients with limb injuries received immobilization that met predefined quality criteria, and overall immobilization quality was strongly associated with EMS personnel's education level.[4] These findings support the need for targeted education on injury assessment and immobilization techniques for EMS professionals and the broader interprofessional trauma team.

Anatomy and Physiology

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Anatomy and Physiology

Musculoskeletal trauma involves injury to 1 or more structures essential to joint stability and function: bone, articular cartilage, synovium, ligament, tendon, joint capsule, and periarticular soft tissues. A joint is the junction where 2 or more bones articulate, allowing controlled motion while maintaining stability. Synovial joints are the most commonly injured joints and are characterized by articular cartilage covering the bone ends, a synovial membrane that produces lubricating synovial fluid, and a capsuloligamentous complex that constrains translation and rotation under physiologic load.[5][6] The surrounding musculotendinous structures provide dynamic stabilization, help distribute joint forces, and influence joint displacement patterns following injury.

The most common orthopedic joint injuries consist of sprains, subluxations, dislocations, fracture-dislocations, and fracture-subluxations. Sprains result from the tearing of 1 or more ligaments, leading to partial or complete disruption of ligament fibers. Subluxations represent a partial loss of articular congruity between joint surfaces. Some degree of contact between the articular surfaces remains. Dislocations are a complete disruption of the joint with total loss of contact between the articular surfaces of the bones. This indicates a gross failure of both static and dynamic stabilizers. These injuries frequently coexist with capsular tears, chondral injury, or fractures, and the risk of neurovascular compromise increases with the degree of displacement.[7]

The physiologic rationale for joint immobilization is to limit pathologic motion across disrupted tissues. Immobilization reduces hemorrhage and prevents progressive ligamentous tearing, chondral shearing, or displacement of associated fractures. Stabilizing the joint also protects adjacent neurovascular structures from traction, compression, or entrapment during movement or transport.[3][4][8]

The shoulder is the most commonly dislocated major joint in the human body. Traumatic glenohumeral dislocations are particularly important for prehospital healthcare professionals to recognize because of their high incidence, distinctive clinical presentation, and significant risk of recurrent instability. Large epidemiologic reviews consistently show that approximately 95% of shoulder dislocations are anterior, resulting from a mechanism that forces the humeral head anteriorly while the arm is abducted and externally rotated. This displacement disrupts the capsulolabral complex, producing the classic deformity of a squared-off shoulder with loss of normal contour, as well as the arm abducted and externally rotated.[9][10] 

Recurrent instability is a major clinical concern, particularly among younger and athletic populations. Results from long-term studies indicate that 40% to 60% of patients experience recurrent dislocations within 2 years, with even higher recurrence rates in patients younger than 25.[11] The axillary nerve is the most frequently affected in shoulder dislocation. Although most neuropraxias are transient, approximately 3% to 6% of cases are associated with persistent sensory changes in the lateral deltoid region, with the risk increased when the dislocation is caused by high-energy trauma or when significant traction is applied during reduction.[12]

Hip dislocations are less common but serious injuries that demand rapid recognition and stabilization. Traumatic hip dislocations occur when the femoral head is displaced from the acetabulum, typically resulting from high-energy mechanisms such as motor vehicle collisions or falls from height. Approximately 90% of traumatic hip dislocations are posterior, presenting with the classic posture of hip flexion, adduction, and internal rotation. Anterior hip dislocations typically present with abduction, flexion, and external rotation of the lower extremity. Damage to the delicate arterial blood supply to the femoral head in these injuries places patients at substantial risk for avascular necrosis (AVN), particularly if reduction is delayed.[13] Significantly increased rates of AVN and posttraumatic arthritis occur when reduction is performed more than 6 hours from injury, underscoring the urgency of early diagnosis, reduction, and stabilization.[14]

Knee dislocations also represent high-risk injuries that require immediate attention. These injuries typically result from violent, high-energy forces such as motor vehicle collisions, though low-energy mechanisms may be responsible in patients with ligamentous laxity or morbid obesity. Approximately 50% of knee dislocations spontaneously reduce before medical attention is sought, making clinical recognition challenging.[15] On examination, continued dislocation, deformity, gross instability, swelling, and difficulty with weight-bearing may be evident. The direction of tibial displacement relative to the femur is used to classify knee dislocations. The finding of an anteriomedial skin furrow over the knee, known as the “dimple sign," suggests a posterolateral dislocation, a subtype of knee dislocation that is irreducible due to soft tissue interposition. Attempts at closed reduction of these dislocations risk skin necrosis and further injury to vessels or nerves.[16][17]

Because the popliteal artery and common peroneal nerve cross the knee joint, knee dislocations pose a significant threat to limb viability. Vascular injury occurs in up to 32% of cases, and delays in revascularization dramatically increase the likelihood of amputation. Peroneal nerve injury occurs in 16% to 40% of patients and is a major predictor of long-term disability, including foot drop. Associated fractures accompany nearly 60% of knee dislocations, further complicating management. In the presence of neurovascular compromise, rapid longitudinal traction and reduction are required, followed by immobilization in slight flexion. Emergent vascular and orthopedic evaluations are vital for optimal care of these patients.[15]

Fracture–dislocations represent high-risk musculoskeletal emergencies that require rapid recognition and careful prehospital management to prevent secondary neurovascular and soft-tissue injury. Current recommendations support that reduction should not be attempted in the field unless there is clear evidence of limb-threatening ischemia, such as absent distal pulses or signs of critical vascular compromise. In these rare situations, gentle, sustained longitudinal traction may be used to restore perfusion; however, only a single attempt, without significant force, should be made to avoid worsening displacement or soft-tissue entrapment. In all other cases, fracture–dislocations should be immobilized in a comfortable position without manipulation and transported to the nearest medical facility.[3][18][19]

Indications

Prehospital immobilization is indicated whenever a patient exhibits signs or symptoms suggestive of underlying bony, ligamentous, or joint injury, including sprains, subluxations, dislocations, and fracture–dislocations. The purpose of immobilization is not simply to support painful extremities but to prevent harmful movement across injured structures, limit soft-tissue damage, and reduce the risk of neurovascular compromise during transport. Even minor malalignment or repetitive micromotion can convert a stable fracture pattern into an unstable one. Effective prehospital immobilization stabilizes the injured limb above and below the involved joint, minimizes pain during movement and transport, and serves as a key first step in preserving limb function until definitive assessment can be completed in the emergency department. Results from studies of extremity trauma show that inadequate early stabilization contributes to increased pain, swelling, and secondary soft-tissue injury, underscoring the importance of proper immobilization in the field.[18]

The decision to splint should be made early, after initial history and examination. Areas of deformity, tenderness, and instabilit as well as neurovascular status and mechanism of injury, should be accounted for. Immobilization is essential in patients with multisystem trauma, where distracting injuries or altered mental status may mask significant extremity pathology. Early splinting in these individuals reduces physiologic stress, improves analgesia, and helps prevent missed extremity injuries during subsequent emergency department resuscitation.[3][18]

Contraindications

Immobilization strategies must be adapted when splinting itself risks worsening open wounds, there is significant contamination, or the patient has crush-related soft-tissue compromise. In these cases, emergency medical service healthcare professionals should prioritize hemorrhage control, gentle limb support, and rapid transport over rigid immobilization. Clinical analyses of lower-extremity trauma emphasize that splinting should never impede access to bleeding control or wound care, and over-compression of swollen compartments may precipitate or worsen ischemic injury.[3]

Equipment

A variety of immobilization devices are available to stabilize musculoskeletal injuries in the prehospital setting, each designed to limit motion, reduce pain, and prevent secondary tissue damage.

  • Arm slings are used to immobilize suspected shoulder injuries and are particularly useful in low-energy shoulder injuries, clavicle fractures, proximal humeral fractures, and postreduction stabilization. They provide a pouch for the arm to sit in, with the weight of the extremity distributed to the neck and upper back. This supports the forearm and decreases gravitational pull across the shoulder, clavicle, and proximal humerus, reducing rotational strain on the glenohumeral joint, improving comfort, and limiting secondary soft-tissue injury.[20][21]
  • Shoulder immobilizers are removable hook-and-loop fastener devices that hold the arm in position across the upper abdomen. Securing the arm firmly against the torso limits abduction, external rotation, and forward flexion. Shoulder immobilizers provide greater restriction of motion than a sling. They are easily applied in the field and offer reliable stabilization during transport.[10]
  • Knee immobilizers are removable circumferential devices that encase the knee joint and provide stabilization from the mid-thigh to the proximal ankle. They maintain the knee in extension, limiting shear forces across the joint. They can be beneficial in suspected ligamentous injuries, tibial plateau fractures, or occult multiligamentous injuries, in which swelling and instability may progress during transport.[15][22]
  • Ankle stirrup splints can be quickly applied in the field. These consist of an air-padded wrap with hook-and-loop fastener straps that secure the splint. These are useful when supplies are limited or rapid evacuation is needed. They help minimize inversion/eversion stress across the ankle joint.[23] 
  • Traction splints can be used for a suspected long bone fracture. The splint consists of a metal exoskeleton that runs longitudinally on either side of the affected extremity, with straps between the parallel metal bars that secure the extremity to the splint. The splint extends beyond the length of the extremity and has a belt intended to be secured to the distal end of the limb. The device applies controlled longitudinal traction to reduce muscle spasm, restore limb length, and decrease internal hemorrhage. Traction splints have been shown to improve anatomical alignment and reduce pain compared with nontraction immobilization.[24][25]
  • Air splints are a deflated air bladder applied circumferentially around the injured extremity. Once applied, they are inflated with air pressure, which applies compressive support to immobilize the covered area. They must be inflated cautiously to avoid excessive pressure that may obscure neurovascular examination or significantly raise intracompartmental pressures.[26][27][28]
  • Vacuum splints are a deflated nylon splint filled with foam-like beads. They are applied longitudinally along the injured extremity and strapped circumferentially. The splint is then deflated under negative pressure, allowing the nylon and bead material to conform to the injured extremity and provide rigid immobilization. Vacuum splints provide superior limb molding compared with rigid or pneumatic splints, reducing point pressure and enabling stabilization of complex contours.[28][29]
  • Rigid splints consist of solid materials that are less flexible than other splints. Materials may include metals, wood, plastics, or other rigid materials. When the rigid material is applied, the splint can then be secured to the extremity with the circumferential application of any available bandage, straps, or materials. These splints provide high stability when secured above and below the injured joint, minimizing rotational and bending forces. They can be adapted into various configurations (posterior splints, sugar tong, volar, ulnar gutter, stirrup) depending on anatomical needs and available materials.[30][31]

Technique or Treatment

Immobilization of joint injuries in the prehospital setting relies on a reproducible, stepwise technique that protects life and limb while avoiding iatrogenic harm. After scene safety is confirmed and a primary survey is performed to address airway, breathing, circulation, and major hemorrhage, the injured joint is exposed and inspected for deformity, open wounds, and gross instability.[32] A focused neurovascular examination (pulses, capillary refill, motor function, and sensation distal to the injury) may be performed and documented before any manipulation.[3]

In general, reducing a dislocation before splinting is preferred when it can be performed safely, but expert consensus advises against routine field reduction of dislocations unless evacuation is prolonged and the clinician is specifically trained. For most patients with intact distal perfusion, the priority is gentle positioning to the least painful, low-tension position, followed by immobilization and rapid transport. When a joint or fracture dislocation is severely angulated, and pulses are absent, or cyanosis is present, a careful attempt at longitudinal traction and alignment to restore perfusion is recommended. If resistance is encountered, the limb should be immobilized in the found position and transport expedited.[18] 

A practical technique for joint immobilization in the field includes:

  • Use standard precautions for possible skin contamination depending on the mechanism of injury.
  • Expose the joint and remove jewelry or constrictive items from the limb.
  • Ensure overall hemodynamic stability; in patients meeting trauma activation criteria, do not delay transport. These patients may have a splint applied en route to the hospital once life-threatening issues have been addressed.
  • Assign a team member to manually stabilize the limb above and below the joint throughout examination and splint application.
  • Perform and document a distal neurovascular exam (pulse, capillary refill, motor, sensation, skin color, and temperature) before manipulation.
  • If distal perfusion is absent, make a single gentle alignment attempt; otherwise, immobilize in a comfortable position and avoid repeated manipulation.
  • Select an immobilization device (rigid, vacuum, or traction splint; sling) that fits the joint and spans the bone above and below the injury. Pad all contact points to avoid pressure injury.
  • Secure the splint while an assistant maintains manual stabilization, then reassess and document distal pulses, motor function, and sensation, comparing these findings with those from the pre-splint examination. Any deterioration mandates loosening or repositioning of the splint and urgent reassessment.[18]

Complications

Joint immobilization is a critical component of prehospital musculoskeletal care, but improper application or prolonged immobilization can lead to significant complications. Neurovascular compromise is a primary concern, as splints that are excessively tight or improperly positioned can restrict blood flow and cause tissue ischemia, neurologic injury, or compartment syndrome. Excessive external compression, especially from circumferential devices, may contribute to pressure-related soft-tissue injury. Similarly, improper padding or splint contours can exacerbate pain, cause shear injury, or lead to blistering.

Malalignment may occur when immobilization is performed without adequate realignment or when the device does not span the joints above and below the injury. This can cause further injury displacement, exacerbate soft-tissue trauma, or complicate later reduction attempts. Certain dislocation patterns are associated with increased skin tension and tissue necrosis when manipulation or attempts at forced reduction occur before immobilization. In these cases, immobilization in the found position is safer when resistance is encountered. Inadequate or improperly performed immobilization that allows motion at the injury site may worsen ligamentous or chondral injury, increase bleeding into the joint or surrounding tissues, and prolong pain and swelling. These risks underscore the importance of appropriate device selection, proper technique, and repeated reassessment during transport.[21][30][33][34]

Clinical Significance

Immobilization of joint injuries in the prehospital setting has direct implications for limb salvage, pain control, and overall trauma outcomes. Advanced trauma life support principles prioritize airway, breathing, and circulation, but once life-threatening problems are addressed, early stabilization of suspected joint injuries becomes a key intervention. Prompt splinting of extremity injuries reduces pathologic motion at the injury site, limits hemorrhage, and facilitates safer transport to definitive care.[32]

Joint injuries are clinically challenging because dislocations, fracture-dislocations, and ligamentous disruptions can coexist and may not be readily distinguishable in the field. As a result, consensus guidance recommends treating all significant joint injuries as potentially unstable. A structured approach should be used with the focus on rapid recognition of injury, assessment of distal neurovascular status, gentle realignment only when there is limb-threatening malperfusion, and timely immobilization in a functional position. Proper immobilization decreases secondary soft-tissue and neurovascular damage, minimizes the risk of converting closed injuries to open injuries during movement, and improves conditions for subsequent reduction and fixation.[18]

Immobilization also contributes substantially to pain control. Results from observational and interventional studies of extremity trauma demonstrate that combining early splinting with pharmacologic analgesia reduces pain scores more effectively than medication alone. Adequate pain control and stabilization are associated with improved patient satisfaction, fewer complications during transport, and more efficient emergency department evaluation.[35] Immobilization of joint injuries is not simply a comfort measure but a key prehospital intervention that bridges initial resuscitation with definitive orthopedic care.

Enhancing Healthcare Team Outcomes

Immobilization of joint injuries in the prehospital setting reduces the risk of secondary injury, facilitates safe transport, and prepares patients for definitive surgical or nonoperative care. Evidence from consensus guidelines and observational studies supports early stabilization and splinting of unstable extremity and joint injuries, including in multisystem trauma, as part of a coordinated strategy that prioritizes life-threatening conditions while still protecting limbs and managing pain.[18] Effective immobilization requires understanding the mechanism of injury, performing thorough neurovascular assessments before and after splinting, and selecting appropriately sized devices.

The clinical impact of immobilization depends on coordinated interprofessional teamwork. Emergency medical service clinicians and other first responders conduct initial assessment and splinting; emergency medicine and orthopedic clinicians provide medical oversight, protocol development, and real-time consultation. Nurses perform continuous monitoring, reassessment, and handoffs in the emergency department, and pharmacists design and evaluate safe, evidence-based pain management protocols.[36]

Clear, standardized communication of injury patterns, neurovascular status, splint type, and medications administered enhances patient-centered care, reduces errors, and allows trauma and orthopedic teams to plan timely definitive management. Data from low- and middle-income settings underscore the value of integrating prehospital healthcare professionals into the overall care system, demonstrating reductions in preventable disability and improved limb outcomes.[37]

References


[1]

DiMaggio CJ, Avraham JB, Lee DC, Frangos SG, Wall SP. The Epidemiology of Emergency Department Trauma Discharges in the United States. Academic emergency medicine : official journal of the Society for Academic Emergency Medicine. 2017 Oct:24(10):1244-1256. doi: 10.1111/acem.13223. Epub 2017 Sep 27     [PubMed PMID: 28493608]


[2]

Mock C, Cherian MN. The global burden of musculoskeletal injuries: challenges and solutions. Clinical orthopaedics and related research. 2008 Oct:466(10):2306-16. doi: 10.1007/s11999-008-0416-z. Epub 2008 Aug 5     [PubMed PMID: 18679760]


[3]

Lee C, Porter KM. Prehospital management of lower limb fractures. Emergency medicine journal : EMJ. 2005 Sep:22(9):660-3     [PubMed PMID: 16113195]


[4]

Adib-Hajbaghery M, Maghaminejad F, Rajabi M. Efficacy of prehospital spine and limb immobilization in multiple trauma patients. Trauma monthly. 2014 Aug:19(3):e16610. doi: 10.5812/traumamon.16610. Epub 2014 Aug 1     [PubMed PMID: 25337517]


[5]

Ralphs JR, Benjamin M. The joint capsule: structure, composition, ageing and disease. Journal of anatomy. 1994 Jun:184 ( Pt 3)(Pt 3):503-9     [PubMed PMID: 7928639]

Level 3 (low-level) evidence

[6]

Khan IM, Redman SN, Williams R, Dowthwaite GP, Oldfield SF, Archer CW. The development of synovial joints. Current topics in developmental biology. 2007:79():1-36     [PubMed PMID: 17498545]

Level 3 (low-level) evidence

[7]

Maffulli N, Del Buono A, Oliva F, Giai Via A, Frizziero A, Barazzuol M, Brancaccio P, Freschi M, Galletti S, Lisitano G, Melegati G, Nanni G, Pasta G, Ramponi C, Rizzo D, Testa V, Valent A. Muscle Injuries: A Brief Guide to Classification and Management. Translational medicine @ UniSa. 2015 May-Aug:12():14-8     [PubMed PMID: 26535183]


[8]

Trentzsch H, Goossen K, Prediger B, Schweigkofler U, Hilbert-Carius P, Hanken H, Gümbel D, Hossfeld B, Lier H, Hinck D, Suda AJ, Achatz G, Bieler D. Stop the bleed " - Prehospital bleeding control in patients with multiple and/or severe injuries - A systematic review and clinical practice guideline - A systematic review and clinical practice guideline. European journal of trauma and emergency surgery : official publication of the European Trauma Society. 2025 Feb 5:51(1):92. doi: 10.1007/s00068-024-02726-1. Epub 2025 Feb 5     [PubMed PMID: 39907772]

Level 1 (high-level) evidence

[9]

Cutts S, Prempeh M, Drew S. Anterior shoulder dislocation. Annals of the Royal College of Surgeons of England. 2009 Jan:91(1):2-7. doi: 10.1308/003588409X359123. Epub     [PubMed PMID: 19126329]


[10]

Kavaja L, Lähdeoja T, Malmivaara A, Paavola M. Treatment after traumatic shoulder dislocation: a systematic review with a network meta-analysis. British journal of sports medicine. 2018 Dec:52(23):1498-1506. doi: 10.1136/bjsports-2017-098539. Epub 2018 Jun 23     [PubMed PMID: 29936432]

Level 1 (high-level) evidence

[11]

Olds M, Ellis R, Donaldson K, Parmar P, Kersten P. Risk factors which predispose first-time traumatic anterior shoulder dislocations to recurrent instability in adults: a systematic review and meta-analysis. British journal of sports medicine. 2015 Jul:49(14):913-22. doi: 10.1136/bjsports-2014-094342. Epub 2015 Apr 21     [PubMed PMID: 25900943]

Level 1 (high-level) evidence

[12]

de Laat EA, Visser CP, Coene LN, Pahlplatz PV, Tavy DL. Nerve lesions in primary shoulder dislocations and humeral neck fractures. A prospective clinical and EMG study. The Journal of bone and joint surgery. British volume. 1994 May:76(3):381-3     [PubMed PMID: 8175837]


[13]

Benedick A, Lopas L, Daley E, Jang Y. Traumatic Hip Dislocation: Pediatric and Adult Evaluation and Management. The Journal of the American Academy of Orthopaedic Surgeons. 2024 Jul 15:32(14):637-646. doi: 10.5435/JAAOS-D-23-01013. Epub 2024 May 2     [PubMed PMID: 38713755]


[14]

Kellam P, Ostrum RF. Systematic Review and Meta-Analysis of Avascular Necrosis and Posttraumatic Arthritis After Traumatic Hip Dislocation. Journal of orthopaedic trauma. 2016 Jan:30(1):10-6. doi: 10.1097/BOT.0000000000000419. Epub     [PubMed PMID: 26849386]

Level 1 (high-level) evidence

[15]

Medina O, Arom GA, Yeranosian MG, Petrigliano FA, McAllister DR. Vascular and nerve injury after knee dislocation: a systematic review. Clinical orthopaedics and related research. 2014 Sep:472(9):2621-9. doi: 10.1007/s11999-014-3511-3. Epub     [PubMed PMID: 24554457]

Level 1 (high-level) evidence

[16]

Boyce RH, Singh K, Obremskey WT. Acute Management of Traumatic Knee Dislocations for the Generalist. The Journal of the American Academy of Orthopaedic Surgeons. 2015 Dec:23(12):761-8. doi: 10.5435/JAAOS-D-14-00349. Epub 2015 Oct 22     [PubMed PMID: 26493970]


[17]

Solarino G, Notarnicola A, Maccagnano G, Piazzolla A, Moretti B. Irreducible posterolateral dislocation of the knee: a case report. Joints. 2015 Apr-Jun:3(2):91-6. doi: 10.11138/jts/2015.3.2.091. Epub 2015 Nov 3     [PubMed PMID: 26605258]

Level 3 (low-level) evidence

[18]

Melamed E, Blumenfeld A, Kalmovich B, Kosashvili Y, Lin G, Israel Defense Forces Medical Corps Consensus Group on Prehospital Care of Orthopedic Injuries. Prehospital care of orthopedic injuries. Prehospital and disaster medicine. 2007 Jan-Feb:22(1):22-5     [PubMed PMID: 17484359]

Level 3 (low-level) evidence

[19]

Cuske J. The lost art of splinting. How to properly immobilize extremities & manage pain. JEMS : a journal of emergency medical services. 2008 Jul:33(7):50-64; quiz 66. doi: 10.1016/S0197-2510(08)70253-5. Epub     [PubMed PMID: 18602591]


[20]

Braun C, McRobert CJ. Conservative management following closed reduction of traumatic anterior dislocation of the shoulder. The Cochrane database of systematic reviews. 2019 May 10:5(5):CD004962. doi: 10.1002/14651858.CD004962.pub4. Epub 2019 May 10     [PubMed PMID: 31074847]

Level 1 (high-level) evidence

[21]

Fennelly JT, Gourbault L, Neal-Smith G, Pradhan A, Gade V, Baxter JA. A systematic review of pre-hospital shoulder reduction techniques for anterior shoulder dislocation and the effect on patient return to function. Chinese journal of traumatology = Zhonghua chuang shang za zhi. 2020 Oct:23(5):295-301. doi: 10.1016/j.cjtee.2020.08.003. Epub 2020 Aug 12     [PubMed PMID: 32893114]

Level 1 (high-level) evidence

[22]

Davenport M, Oczypok MP. Knee and Leg Injuries. Emergency medicine clinics of North America. 2020 Feb:38(1):143-165. doi: 10.1016/j.emc.2019.09.012. Epub     [PubMed PMID: 31757247]


[23]

Leanderson J, Wredmark T. Treatment of acute ankle sprain. Comparison of a semi-rigid ankle brace and compression bandage in 73 patients. Acta orthopaedica Scandinavica. 1995 Dec:66(6):529-31     [PubMed PMID: 8553821]


[24]

Philipsen SPJ, Vergunst AA, Tan ECTH. Traction Splinting for midshaft femoral fractures in the pre-hospital and Emergency Department environment-A systematic review. Injury. 2022 Dec:53(12):4129-4138. doi: 10.1016/j.injury.2022.09.051. Epub 2022 Sep 27     [PubMed PMID: 36229245]

Level 1 (high-level) evidence

[25]

Davis DD, Ginglen JG, Kwon YH, Kahwaji CI. EMS Traction Splint. StatPearls. 2025 Jan:():     [PubMed PMID: 29939619]


[26]

Hurt HF, Reilly AJ. Improvised Hydration Bladder Air Splint: A Wilderness Case Report. Wilderness & environmental medicine. 2019 Mar:30(1):86-89. doi: 10.1016/j.wem.2018.10.008. Epub 2019 Jan 11     [PubMed PMID: 30642710]

Level 3 (low-level) evidence

[27]

Ashton H. Effect of inflatable plastic splints on blood flow. British medical journal. 1966 Dec 10:2(5527):1427-30     [PubMed PMID: 5927673]


[28]

Powell RA, Weir AJ. EMS Bone Immobilization. StatPearls. 2025 Jan:():     [PubMed PMID: 29939555]


[29]

Letts RM, Hobson DA. The vacuum splint: an aid in emergency splinting of fractures. Canadian Medical Association journal. 1973 Oct 6:109(7):599-600     [PubMed PMID: 4742489]


[30]

Collopy KT, Kivlehan SM, Snyder SR. Managing unstable musculoskeletal injuries: EMS world. 2012 Feb:41(2):36-43     [PubMed PMID: 22413699]


[31]

Perkins TJ. Fracture management. Effective prehospital splinting techniques. Emergency medical services. 2007 Apr:36(4):35-7, 39     [PubMed PMID: 17461378]


[32]

Worsing RA Jr. Principles of prehospital care of musculoskeletal injuries. Emergency medicine clinics of North America. 1984 May:2(2):205-17     [PubMed PMID: 6394300]


[33]

Pernik MN, Seidel HH, Blalock RE, Burgess AR, Horodyski M, Rechtine GR, Prasarn ML. Comparison of tissue-interface pressure in healthy subjects lying on two trauma splinting devices: The vacuum mattress splint and long spine board. Injury. 2016 Aug:47(8):1801-5. doi: 10.1016/j.injury.2016.05.018. Epub 2016 Jun 4     [PubMed PMID: 27324323]


[34]

Wood SP, Vrahas M, Wedel SK. Femur fracture immobilization with traction splints in multisystem trauma patients. Prehospital emergency care. 2003 Apr-Jun:7(2):241-3     [PubMed PMID: 12710786]


[35]

Abebe Y, Hetmann F, Sumera K, Holland M, Staff T. The effectiveness and safety of paediatric prehospital pain management: a systematic review. Scandinavian journal of trauma, resuscitation and emergency medicine. 2021 Dec 11:29(1):170. doi: 10.1186/s13049-021-00974-3. Epub 2021 Dec 11     [PubMed PMID: 34895311]

Level 1 (high-level) evidence

[36]

Čretnik A, Pfeifer R. 5. Prehospital management. European journal of trauma and emergency surgery : official publication of the European Trauma Society. 2025 May 6:51(1):198. doi: 10.1007/s00068-025-02825-7. Epub 2025 May 6     [PubMed PMID: 40329092]


[37]

Unadkat A, Stoller E, Pine H, Eisner ZJ, Klapow MC, Kulkarni AJ, Thiagarajan A, Smith N, Delaney PG. Prehospital Extremity Fracture Management in Low and Middle-Income Countries: A Scoping Review of Lay First Responders and Traditional Bonesetters. World journal of surgery. 2025 Aug:49(8):2255-2263. doi: 10.1002/wjs.12678. Epub 2025 Jul 11     [PubMed PMID: 40646650]

Level 2 (mid-level) evidence